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Figure 1 Table 1 Major metabolic abnormalities in myocardial hypertrophy and biomarker implications
A plausible explanation could be a reorientation of Lys105 towards GS resulting in a decreased stabilization of the active site thiolate as a leaving group
Patients should be reassured that most treatment-related fatigue is self-limiting
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This dual mechanism of action is significant because it not only enhances insulin secretion in response to high blood glucose levels but also reduces glucagon secretion and delays gastric emptying which can contribute to weight loss
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